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A Fresh Perspective from Clinical Experience
High on the list of possible triggers for carpal tunnel syndrome (CTS) are blood sugar imbalances and metabolic issues.
However, it still is generally accepted that carpal tunnel syndrome is caused exclusively by repeat motion injuries, extreme tension, rheumatoid arthritis, or neuropathy (a complication of diabetes).
From years of gripping, pulling, twisting or typing action, CTS in these cases is the result of the narrowing of the nerve duct at the base of the wrist. This narrowing and constant pressure exerted on the sheath of the nerve may lead to inflammation and to temporary or permanent nerve damage, extreme pain, and reduced range of motion.
Common mainstream remedial approaches include corticosteroids, insulin injections, and eventually surgery; latter with a lengthy list of research dealing with surgery side effects. Treatment with corticosteroids in itself bears an increased risk of diabetes as a corticosteroid side effect.
For more than a quarter of a century I have been working with patients who looked me up for help with constant pain in both wrists or individuals who came to see me after they had undergone bilateral surgery for carpal tunnel syndrome. It is remarkable that many of these individuals kept complaining about continued and unchanged pain long after their CTS surgery. From my clinical experience—and, as a former pianist—I can state that it is rather unusual to find repeat motion triggered carpal tunnel syndrome simultaneously in both wrists.
But, this is precisely what we are seeing in many a diabetic or pre-diabetic person. Initially, a small 2006 English study found that at least thirty-five percent of those individuals diagnosed with carpal tunnel syndrome might already be affected by metabolic changes. In plain English, these individuals found out that they were pre-diabetic when they were diagnosed with carpal tunnel syndrome.
Since then, mainstream research identifies CTS as among the early risk indicators and warming signs for possible pre-diabetes or diabetes. Vice versa, many diabetics develop signs and symptoms of CTS.
Although a lot of research already points to close links between CTS and metabolic disease, a first line response to resolve CTS by controlling, preventing or reversing metabolic issues naturally rarely enters the clinically minded mainstream approach. But in many patients surgery could be avoided.
The nerve damage leading to carpal tunnel syndrome in many individuals may quite easily be explained by the known damage of high blood sugar levels on the fine nerve endings, particularly those of your extremities. Most importantly, these findings confirm that a lot of physical and functional damage occurs to the body well before the individual becomes aware that they are showing signs of pre-diabetes or even full-blown diabetes.
The earlier one recognizes a possible risk of underlying metabolic disorders the more can be done to counter it. In my book "At Risk? Avoid Diabetes by Recognizing Early Risk – A Natural Medicine Approach" I describe and cite over fifty conditions identified by research as early diabetes risk indicators.