Visit our Re-post guidelines
Fluoride is put in your drinking water 'for your teeth' without your consent, but did you know that it could also be calcifying your arteries?
A few years ago, we reported on a study evaluating a new diagnostic technology that inadvertently revealed a link between fluoride exposure and coronary artery disease. Our report stirred up quite a lot of controversy and criticism, even leading one of the most respected figures in alternative medicine (deservedly so) – Dr. Russell Blaylock -- to call us out on Infowars for our allegedly sophomoric interpretation of the following article: "Association of vascular fluoride uptake with vascular calcification and coronary artery disease." As one can see, the study's results revealed a hitherto largely unknown connection between fluoride exposure, coronary artery disease and cardiovascular events (e.g. heart attack).
"There was significant correlation between history of cardiovascular events and presence of fluoride uptake in coronary arteries. The coronary fluoride uptake value in patients with cardiovascular events was significantly higher than in patients without cardiovascular events."
The argument, at the time, was the study was simply about a new diagnostic technique and shouldn't be 'read into,' and that, presumably, the increased fluoride uptake value observed in patients with a higher frequency of cardiovascular events was a an 'effect' of the heart disease itself and not in any way indicative of fluoride's causative role as a cardiotoxic agent -- despite the fact that fluoride's cardiotoxicity has already been consistently demonstrated in the biomedical literature.
Now, a provocative new study published in the journal Toxicology not only provides some vindication for our previous interpretations, but also raises serious concern over the cardiovascular complications associated with water fluoridation practices, showing for the first time that despite exhibiting an anti-calcification effect in vitro (cell model) fluoride exposure at levels found in people who drink fluoridated water exhibits artery-calcifying effects in the more important in vivo (animal) model.
Titled, "Effect of water fluoridation on the development of medial vascular calcification in uremic rats," the study opens with a description of the common medical justification for public water fluoridation:
"In order to improve dental health in the population, fluoride is included in tooth pastes and mouthwash solutions or is added to public water supplies at 0.5–1.5 mg/L (WHO, 2008), which has been a common practice in some countries since 1945."
And yet, the study acknowledges that fluoride is a well-established toxicant that our body has to either incorporate into its tissues or excrete through the kidney's to sequester or eliminate:
"More than 90% of ingested fluoride is absorbed through the intestine and quickly distributed between plasma/soft tissues and calcified structures, where it can be sequestered for years (Buzalaf and Whitford, 2011). When water is fluoridated at the WHO- recommended levels, the range of plasma fluoride concentration is usually 1–6 uM (Husdan et al., 1976; Singer and Ophaug, 1979). Fluoride is not under homeostatic control, and it is cleared from the plasma within few hours by the complementary action of calcified tissues and the kidneys."
Those with chronic kidney disease have a harder time clearing the fluoride, which results in increased blood plasma levels, especially as the length of exposure increases.
The study noted that in healthy people, almost without exception, fluoride accumulates in the aorta, and in the elderly can exceed 100 ug/g [microgram/gram] tissue. Since atherosclerosis involves the gradual hardening and final calcification of the arteries with a form of calcium known as hydroxylapatite, fluoride's role in replacing hydroxyls within hydroxylapatite crystals to form fluorapatite can be considered enhancing the cardiotoxicity of these calcium deposits due to the fact that fluorapatite is less soluble than hydroxylapatite and therefore more resistant to the body's demineralization mechanisms (or de-calcification with natural substances such as magnesium, hawthorn or vitamin K2). The authors address this point:
"From a therapeutic point of view, this incorporation [of fluoride into hydroxylapatite as fluorapatite] may involve an additional problem, because these calcifications will be more difficult to eliminate, if at all possible."
The report discussed how despite the observation that fluoride accumulates in the main arteries, "the effects on the vascular wall are not clear." A brief review of the literature shows highly contradictory results, with some studies implying fluoride exposure actually reduces aortic calcification and others showing (as would be expected) deleterious effects on the cardiovascular system. This uncertainty was one of the main reasons they designed their study: