New Study Signals Wheat-Autism Link

New Study Confirms Wheat-Autism Link

As the autism epidemic continues to accelerate, one of the least well known contributing factors goes mostly unnoticed: wheat consumption. 

A new study published in the open access journal PLoS is reinvigorating the debate over what are the primary causes of the accelerating autism epidemic. Whereas too many within the conventional medical establishment, failing to identify a singular cause, apathetically label the condition "idiopathic," i.e. cause unknown, or worse, "caused by unknown defective genes," there is a growing awareness that a multitude of environmental factors including vaccines, chemical exposures, C-sections, antibiotics, genetically modified food, and food intolerances are essential in both understanding and treating this disturbing affliction.

The point therefore is not to prove one thing the cause (e.g. "autism genes"), and another not the cause (e.g. vaccines), as if it were strictly some kind of academic sport or past time, rather, to acknowledge possible contributing factors, and eliminate them whenever possible as a precaution.

The new study titled, "Markers of Celiac Disease and Gluten Sensitivity in Children with Autism," explored the possible association between gluten sensitivity and autism, which previous research has confirmed only inconsistently.[i]

They described their aim as follows: "[T]o assess immune reactivity to gluten in pediatric patients diagnosed with autism according to strict criteria and to evaluate the potential link between autism and celiac disease."

They discovered that children with autism had significantly higher levels of IgG antibodies to gliadin, the primary immunotoxic protein in wheat, compared with healthy controls. They also found that the IgG anti-gliadin antibody response was significantly greater in the autistic children with gastrointestinal symptoms in comparison to those without them. The elevated IgG antibodies indicate: 1) that their immune systems are identifying gliadin proteins as "other." 2) that gliadin is not being properly broken down through digestive processes and are entering through a compromised intestinal tract ("leaky gut") into the blood.

It has been theorized that when the immune system forms antibodies against gliadin, these antibodies cross react with self-structures within the nervous system. Known as molecular mimicry, this break down of immunological self-tolerance can contribute to wide range of neurological problems including neuropathy, ataxia, seizures, and neurobehavioral changes including mania, schizophrenia and autism. Anti-gliadin antibodies are therefore a possible cause of autoimmune neurological damage.  One study published in the Journal of Immunology in 2007 found that anti-gliadin antibodies crossreact with the neurological protein class known as synapsin I, providing a molecular mechanism behind how an immune reaction to gliadin could contribute to neurological degeneration.

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Chart of increase in autism: perhaps vitamin D



 There are a great many studies showing a strong association between autism and low levels of vitamin D.  Also there is a great video in June 2013 which summarizes the current information. It gives 27 reasons to believe that autism is linked to low vitamin D, and may be able to treat it as well.

Details at http://is.gd/DAutism

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