Anemia: Hemolytic https://greenmedinfo.com/taxonomy/term/2452/all en Argania spinosa has antioxidant properties. https://greenmedinfo.com/article/argania-spinosa-has-antioxidant-properties PMID:  Fitoterapia. 2008 Jul ;79(5):337-44. Epub 2008 Apr 18. PMID: 18514434 Abstract Title:  Protective effect of saponins from Argania spinosa against free radical-induced oxidative haemolysis. Abstract:  Saponins from Argania spinosa at a non-haemolytic concentration diminish by 53.2% erythrocyte haemolysis induced by free radicals. 2 mM aspirin and acetaminophen diminish by 75% and 68% , respectively, erythrocyte haemolysis induced by free radicals, while 0.3 microM vitamin E shows no significant antioxidant activity. Interestingly, a combination of 1 mg/l of A. spinosa saponins and vitamin E at 0.3 microM resulted in a 68% level of protection against free radical-induced erythrocyte haemolysis, which may suggest that A. spinosa saponins enhance the antioxidant effect of vitamin E. In contrast, no synergic effect was observed for acetaminophen (2 mM) when in combination with vitamin E (0.3 microM). These results demonstrate the antioxidant properties of saponins from A. spinosa and their ability to potentate the antioxidant effect of vitamin E. https://greenmedinfo.com/article/argania-spinosa-has-antioxidant-properties#comments Anemia: Hemolytic Argania spinosa (Argan) Oxidative Stress Antioxidants In Vitro Study Mon, 18 Feb 2013 17:56:32 +0000 greenmedinfo 91403 at https://greenmedinfo.com Aspirin (Acetylsalicylic acid) may cause severe hemolytic anemia in some glucose-6-phosphate-dehydrogenase-deficient (G-6-PD-deficient) individuals. https://greenmedinfo.com/article/aspirin-acetylsalicylic-acid-may-cause-severe-hemolytic-anemia-some-glucose-6- Acetylsalicylic acid (ASA) is known to cause severe hemolytic anemia in some glucose-6-phosphate-dehydrogenase-deficient (G-6-PD-deficient) individuals. To study its mechanism, erythrocytes from an ASA-sensitive patient were transfused into a normal compatible recipient. The administration of 2,5-dihydroxybenzoic (gentisic) acid, a known ASA metabolite with redox properties, to the recipient resulted in a marked decrease in the survival of the patient&#039;s erythrocytes. Similar studies with red cells from individuals with A- and Mediterranean variants of G-6-PD revealed no alteration in the erythrocytes&#039; survival. Further studies disclosed that both salicylate and gentisate competitively inhibited the G-6-PD from the ASA-sensitive patient resulting in a marked change in the K(m) for NADP. These drugs also inhibited the A- and Mediterranean variants of G-6-PD. The magnitude of inhibition, however, was comparatively small and not different from that observed with a normal enzyme. The above studies suggested that enzyme inhibition by salicylate and gentisate may play an important role in ASA-induced hemolysis. Such an inhibition would further curtail NADPH regeneration, rendering the cells more vulnerable to oxidants. In this connection, gentisate seems to play a major role in ASA-induced hemolysis for it is both a G-6-PD inhibitor and an &quot;oxidant.&quot; PMID:  J Clin Invest. 1970 Jul;49(7):1334-40. PMID: 5432368 Abstract Title:  Acetylsalicylic acid--induced hemolysis and its mechanism. Abstract:  Acetylsalicylic acid (ASA) is known to cause severe hemolytic anemia in some glucose-6-phosphate-dehydrogenase-deficient (G-6-PD-deficient) individuals. To study its mechanism, erythrocytes from an ASA-sensitive patient were transfused into a normal compatible recipient. The administration of 2,5-dihydroxybenzoic (gentisic) acid, a known ASA metabolite with redox properties, to the recipient resulted in a marked decrease in the survival of the patient&#039;s erythrocytes. Similar studies with red cells from individuals with A- and Mediterranean variants of G-6-PD revealed no alteration in the erythrocytes&#039; survival. Further studies disclosed that both salicylate and gentisate competitively inhibited the G-6-PD from the ASA-sensitive patient resulting in a marked change in the K(m) for NADP. These drugs also inhibited the A- and Mediterranean variants of G-6-PD. The magnitude of inhibition, however, was comparatively small and not different from that observed with a normal enzyme. The above studies suggested that enzyme inhibition by salicylate and gentisate may play an important role in ASA-induced hemolysis. Such an inhibition would further curtail NADPH regeneration, rendering the cells more vulnerable to oxidants. In this connection, gentisate seems to play a major role in ASA-induced hemolysis for it is both a G-6-PD inhibitor and an &quot;oxidant.&quot; <p><a href="https://greenmedinfo.com/article/aspirin-acetylsalicylic-acid-may-cause-severe-hemolytic-anemia-some-glucose-6-" target="_blank">read more</a></p> https://greenmedinfo.com/article/aspirin-acetylsalicylic-acid-may-cause-severe-hemolytic-anemia-some-glucose-6-#comments Anemia: Hemolytic Glucose-6-phosphate-dehydrogenase-deficient (G-6-PD-deficient) Glucosephosphate Dehydrogenase Deficiency Aspirin Hemolytic Human Study Sat, 18 Sep 2010 18:37:03 +0000 greenmedinfo 56780 at https://greenmedinfo.com Autoimmume hemolytic anemia and Epstein-Barr virus infection may be causatively linked. https://greenmedinfo.com/article/autoimmume-hemolytic-anemia-and-epstein-barr-virus-infection-may-be-causativel PMID:  Intern Med. 2010;49(4):325-9. Epub 2010 Feb 15. PMID: 20154439 Abstract Title:  Autoimmune hemolytic anemia accompanied by reactivation of an Epstein-Barr virus infection with suppressed CTL response to EBV-infected cells in an elderly man. Abstract:  An 88-year-old man with autoimmune hemolytic anemia (AIHA) who had been treated with low dose prednisolone developed a sudden worsening of his anemia accompanied by reactivation of Epstein-Barr virus (EBV). We established EBV-infected spontaneous lymphoblastoid cell lines (LCL), performed an enzyme-linked immunosorbent spot assay, and confirmed a significantly suppressed EBV-specific cytotoxic T-cell (CTL) response to the LCL. EBV reactivation might have been brought about by suppressed CTL activity which could have been due to low dose PSL administration or aging. Since the EBV-DNA titer decreased as AIHA improved, we concluded that EBV might have played a role in the development of anemia. https://greenmedinfo.com/article/autoimmume-hemolytic-anemia-and-epstein-barr-virus-infection-may-be-causativel#comments Anemia: Hemolytic Epstein-Barr Virus Infections Diseases that are Linked Human Study Fri, 19 Feb 2010 19:58:16 +0000 greenmedinfo 51892 at https://greenmedinfo.com Harmful algal toxins from Florida red tide can cause a wide range of adverse health effects. https://greenmedinfo.com/article/harmful-algal-toxins-florida-red-tide-can-cause-wide-range-adverse-health-effe PMID:  Ecotoxicology. 2008 Oct ;17(7):623-31. Epub 2008 Aug 30. PMID: 18758951 Abstract Title:  Harmful algal toxins of the Florida red tide (Karenia brevis): natural chemical stressors in South Florida coastal ecosystems. Abstract:  The Florida red tide is a descriptive name for high concentrations of the harmful marine alga, Karenia brevis. Although most prevalent along the south-west Florida coast, periodic blooms have occurred throughout the entire US and Mexico Gulf coasts and the Atlantic coast to North Carolina. This dinoflagellate produces a suite of polyether neurotoxins, called brevetoxins, that cause severe impacts to natural resources, as well as public health. These naturally produced biotoxins may represent one of the most common chemical stressors impacting South Florida coastal and marine ecosystems. Impacts include massive fish kills, marine mammal, sea turtle and sea bird mortalities, benthic community die-off and public health effects from shellfish contamination and inhalation of air-borne toxins. The primary mode of action is binding to voltage-gated sodium channels causing depolarization of nerve cells, thus interfering with nerve transmission. Other effects include immune depression, bronchial constriction and haemolysis. Parent algal toxins are synthesized within the unicellular organism, others are produced as metabolic products. Recent studies into the composition of brevetoxins in cells, water, air and organisms have shown PbTx-2 to be the primary intracellular brevetoxin that is converted over time to PbTx-3 when the cells are ruptured, releasing extracellular brevetoxins into the environment. Brevetoxins become aerosolized by bubble-mediated transport of extracellular toxins, the composition of which varies depending on the composition in the source water. Bivalved molluscs rapidly accumulate brevetoxins as they filter feed on K. brevis cells. However, the parent algal toxins are rapidly metabolized to other compounds, some of which are responsible for neurotoxic shellfish poisoning (NSP). These results provide new insight into the distribution, persistence and impacts of red tide toxins to south-west Florida ecosystems. https://greenmedinfo.com/article/harmful-algal-toxins-florida-red-tide-can-cause-wide-range-adverse-health-effe#comments Anemia: Hemolytic Asthma Immune Disorders: Low Immune Function Red Tide Associated Toxicity Respiratory Diseases Brevetoxin Bronchoconstriction Immunosuppressive Red tide Review Thu, 08 Dec 2011 20:36:34 +0000 greenmedinfo 70173 at https://greenmedinfo.com Hemolytic Anemia https://greenmedinfo.com/disease/hemolytic-anemia <div class="field field-image"> <div class="field-items"> <div class="field-item odd"> <img class="imagefield imagefield-field_image" width="450" height="248" alt="" src="//cdn.greenmedinfo.com/sites/default/files/HemolyticAnemia.jpg?1477256331" /> </div> </div> </div> <div class="field field-copyright"> <div class="field-items"> <div class="field-item odd"> Copyright: &lt;a href=&#039;http://www.123rf.com/profile_joshyabb&#039;&gt;joshyabb / 123RF Stock Photo&lt;/a&gt; </div> </div> </div> <fieldset class="fieldgroup group-facebook-like-info"><legend>Facebook Like Info</legend><div class="field field-facebook-total-count"> <div class="field-items"> <div class="field-item odd"> 0 </div> </div> </div> </fieldset> Anemia: Hemolytic Tue, 14 Apr 2009 07:19:05 +0000 greenmedinfo 20299 at https://greenmedinfo.com High-dose vitamins E and C supplementation prevents ribavirin-induced hemolytic anemia in patients with chronic hepatitis C. https://greenmedinfo.com/article/high-dose-vitamins-e-and-c-supplementation-prevents-ribavirin-induced-hemolyti PMID:  Hepatol Res. 2007 May;37(5):317-24. PMID: 17441803 Abstract Title:  High-dose vitamins E and C supplementation prevents ribavirin-induced hemolytic anemia in patients with chronic hepatitis C. Abstract:  Aim: In combination therapy using interferon (IFN) and ribavirin for chronic hepatitis C, reduced doses should be used due to ribavirin-induced hemolytic anemia. The present study aimed to elucidate whether high-dose vitamins E and C supplementation attenuated ribavirin-induced hemolytic anemia. Methods: Twenty-one consecutive patients with chronic hepatitis C were enrolled in this study between July 2003 and December 2004, and received high-dose vitamins E (2000 mg) and C (2000 mg) supplementation, daily, in addition to IFN alfa-2b and ribavirin combination therapy (vitamins E/C group). Twenty-one sex- and age-matched patients who received a standard regimen of IFN alfa-2b and ribavirin for chronic hepatitis C between January 2001 and June 2003 were evaluated as the control group. Results: Decrease in hemoglobin level was significantly prevented in the vitamins E and C group compared to that in the control group (P = 0.029). Three (14.3%) patients in the control group discontinued treatment because of anemia, while no treated patient dropped out of the study due to anemia. Sustained virological response was not significantly different between the two groups. Conclusion: High-dose vitamins E and C supplementation prevented ribavirin-induced hemolytic anemia during combination therapy with ribavirin and IFN alfa-2b in patients with chronic hepatitis C. https://greenmedinfo.com/article/high-dose-vitamins-e-and-c-supplementation-prevents-ribavirin-induced-hemolyti#comments Anemia: Hemolytic Hepatitis C Vitamin C Vitamin E Drug Side Effect Attenuation Human Study Fri, 01 Jan 2010 14:13:18 +0000 greenmedinfo 48537 at https://greenmedinfo.com Jaundice: Hemolytic https://greenmedinfo.com/disease/jaundice-hemolytic <div class="field field-image"> <div class="field-items"> <div class="field-item odd"> <img class="imagefield imagefield-field_image" width="450" height="300" alt="" src="//cdn.greenmedinfo.com/sites/default/files/JaundiceBaby_3.jpg?1478816624" /> </div> </div> </div> <div class="field field-copyright"> <div class="field-items"> <div class="field-item odd"> Copyright: &lt;a href=&#039;http://www.123rf.com/profile_traida&#039;&gt;traida / 123RF Stock Photo&lt;/a&gt; </div> </div> </div> <fieldset class="fieldgroup group-facebook-like-info"><legend>Facebook Like Info</legend><div class="field field-facebook-total-count"> <div class="field-items"> <div class="field-item odd"> 0 </div> </div> </div> </fieldset> Anemia: Hemolytic Tue, 14 Apr 2009 07:20:34 +0000 greenmedinfo 20662 at https://greenmedinfo.com Magnesium deficiency may be associated with hemolytic anemia. https://greenmedinfo.com/article/magnesium-deficiency-may-be-associated-hemolytic-anemia PMID:  Blood. 1973 Mar;41(3):451-9. PMID: 4690142 Abstract Title:  The hemolytic anemia of magnesium deficiency in adult rats. Abstract:  A magnesium-deficient diet given to rats for 4 to 5 weeks resulted in anemia. The anemia that is produced by magnesium deficiency is a hemolytic type that results from a combination of a reversible extrinsic defect and an irreversible structural defect in the red blood cell. https://greenmedinfo.com/article/magnesium-deficiency-may-be-associated-hemolytic-anemia#comments Anemia: Hemolytic Magnesium Magnesium Deficiency Animal Study Fri, 19 Feb 2010 21:06:27 +0000 greenmedinfo 51893 at https://greenmedinfo.com Polymeric (polystyrene) nano particles have hemolytic properties. https://greenmedinfo.com/article/polymeric-polystyrene-nano-particles-have-hemolytic-properties PMID:  IEEE Trans Nanobioscience. 2011 Dec ;10(4):259-61. Epub 2011 Nov 22. PMID: 22128012 Abstract Title:  Hemolytic effect of polymeric nanoparticles: role of albumin. Abstract:  Nanoparticles (NP) have drawn increasing interest from many fields in medicine and are a relatively new class of biomedical products. Because there are concerns about the health effects of nanoparticles, it is important to understand how nanoparticles interact specifically with red blood cells (RBC), a central object in the blood circulation. As numerous studies that have examined NP/RBC interaction concentrated on the hemolytic potential of nanoparticles, we describe an investigation of hemolytic activity of polystyrene nanoparticles (PS-NP) in protein free medium and its modulation by albumin. We found that treatment of RBCs with PS-NP induces hemolysis (dose and particle size dependent) in plasma free medium but not in full plasma or in buffer, which contain albumin. Critical albumin concentration is 0.05% wt. According to our results hemolytic effect of nanoparticles is strongly modulated by protein concentration in the medium. https://greenmedinfo.com/article/polymeric-polystyrene-nano-particles-have-hemolytic-properties#comments Anemia: Hemolytic Blood Diseases Hemolytic Nanotechnology Polystyrene nanoparticles In Vitro Study Sat, 14 Apr 2012 21:41:42 +0000 greenmedinfo 74322 at https://greenmedinfo.com Proton pump inhibitors induce hemolytic anemia. https://greenmedinfo.com/article/proton-pump-inhibitors-induce-hemolytic-anemia PMID:  J Family Med Prim Care. 2019 Apr ;8(4):1486-1487. PMID: 31143745 Abstract Title:  Proton pump inhibitors induce hemolytic anemia. Abstract:  Proton pump inhibitors (PPIs) are generally safe, and their short-term use commonly does not induce hemolytic anemia. The underlying mechanisms are unknown, and the literature insufficiently explores hemolytic anemia as an adverse event induced by PPIs. In this case study, we report a 31-year-old female who had developed hemolytic anemia with symptoms of jaundice, hyperbilirubinemia, and high reticulocytes, after treatment with PPI. Interestingly, the patient completely recovered after PPI withdrawal. This present case study highlights the need for physicians to exercise caution when treating patients with PPI. <p><a href="https://greenmedinfo.com/article/proton-pump-inhibitors-induce-hemolytic-anemia" target="_blank">read more</a></p> https://greenmedinfo.com/article/proton-pump-inhibitors-induce-hemolytic-anemia#comments Anemia: Hemolytic Proton-Pump Inhibitors Increased Risk Human: Case Report Mon, 02 Dec 2019 20:15:33 +0000 greenmedinfo 203482 at https://greenmedinfo.com Pycnogenol prevents haemolytic injury in G6PD deficient human erythrocytes. https://greenmedinfo.com/article/pycnogenol-prevents-haemolytic-injury-g6pd-deficient-human-erythrocytes PMID:  Phytother Res. 2003 Jun;17(6):671-4. PMID: 12820238 Abstract Title:  Pycnogenol prevents haemolytic injury in G6PD deficient human erythrocytes. Abstract:  Glucose6 phosphate dehydrogenase (G6PD) deficiency is the most common X-linked disorder of human erythrocytes where cells have inadequate capacity to destroy peroxides and high susceptibility towards haemolytic changes. Pycnogenol is a proprietary dry extract of the French Maritime pine (Pinus pinaster) bark with high ability to scavenge free radicals. In the present study we have investigated if Pycnogenol can protect G6PD deficient erythrocytes against haemolytic cell damage. Venous blood samples were obtained from six subject of Mediterranean origin with known G6PD deficiency which was also confirmed with standard techniques. Erythrocyte haemolysis in the presence and absence of Pycnogenol was induced either with tert-butylhydroperoxide (t-BHP) or quinine and the haemoglobin release in the supernatant was determined by recording the optical density at 540 nm in a Shimadzu spectrophotometer. Our results have shown that Pycnogenol has protective action against a Xenobiotic chemical induced haemolysis in G6PD deficient human erythrocytes. https://greenmedinfo.com/article/pycnogenol-prevents-haemolytic-injury-g6pd-deficient-human-erythrocytes#comments Anemia: Hemolytic Flavonoids Glucosephosphate Dehydrogenase Deficiency Pycnogenol (Pine Bark) Plant Extracts In Vitro Study Sun, 21 Feb 2010 15:10:15 +0000 greenmedinfo 52199 at https://greenmedinfo.com Stannous chloride (tin) causes hemolytic anemia in rabbits through adversely effecting the hematopoietic system in a manner similar to lead. https://greenmedinfo.com/article/stannous-chloride-tin-causes-hemolytic-anemia-rabbits-through-adversely-effect Because of the importance of stannous chloride in various fields of human endeavour, the potential genotoxicity of this reducing agent was evaluated by measurement of either the inactivation or the induction of SOS responses in bacteria. Escherichia coli strains used in this work (wild type, uvrA, recA, lexA and uvrA recA) were treated with stannous chloride; the wild type was found to be the most resistant and the double mutant, the most sensitive strain. As these strains present mutations on specific genes for the repair of DNA, stannous chloride would appear to be capable of inducing and/or producing lesions in DNA and, thus, can be considered to be a potential genotoxic agent. This capability was confirmed by the lysogenic induction of E. coli K12 (lambda) (Inductest) and by microscopic observation of E. coli B filamentation. PMID:  Food Chem Toxicol. 1994 May;32(5):477-9. PMID: 8206446 Abstract Title:  Evaluation of potential genotoxicity of stannous chloride: inactivation, filamentation and lysogenic induction of Escherichia coli. Abstract:  Because of the importance of stannous chloride in various fields of human endeavour, the potential genotoxicity of this reducing agent was evaluated by measurement of either the inactivation or the induction of SOS responses in bacteria. Escherichia coli strains used in this work (wild type, uvrA, recA, lexA and uvrA recA) were treated with stannous chloride; the wild type was found to be the most resistant and the double mutant, the most sensitive strain. As these strains present mutations on specific genes for the repair of DNA, stannous chloride would appear to be capable of inducing and/or producing lesions in DNA and, thus, can be considered to be a potential genotoxic agent. This capability was confirmed by the lysogenic induction of E. coli K12 (lambda) (Inductest) and by microscopic observation of E. coli B filamentation. <p><a href="https://greenmedinfo.com/article/stannous-chloride-tin-causes-hemolytic-anemia-rabbits-through-adversely-effect" target="_blank">read more</a></p> https://greenmedinfo.com/article/stannous-chloride-tin-causes-hemolytic-anemia-rabbits-through-adversely-effect#comments Anemia: Hemolytic Tin Poisoning Stannous chloride Animal Study Mon, 15 Feb 2010 15:07:11 +0000 greenmedinfo 51463 at https://greenmedinfo.com There is a temporal association between vaccines and hemolytic anemia in dogs. https://greenmedinfo.com/article/there-temporal-association-between-vaccines-and-hemolytic-anemia-dogs PMID:  J Vet Intern Med. 1996 Sep-Oct;10(5):290-5. PMID: 8884713 Abstract Title:  Vaccine-associated immune-mediated hemolytic anemia in the dog. Abstract:  Vaccination has been incriminated as a trigger of immune-mediated hemolytic anemia (IMHA) in dogs and in people, but evidence to support this association is lacking. In a controlled retrospective study, idiopathic IMHA was identified in 58 dogs over a 27-month period. When compared with a randomly selected control group of 70 dogs (presented for reasons other than IMHA) over the same period, the distribution of cases versus time since vaccination was different (P<p><a href="https://greenmedinfo.com/article/there-temporal-association-between-vaccines-and-hemolytic-anemia-dogs" target="_blank">read more</a></p> https://greenmedinfo.com/article/there-temporal-association-between-vaccines-and-hemolytic-anemia-dogs#comments Anemia Anemia: Hemolytic Vaccine-induced Toxicity Hemolytic Vaccination: All Animal Study Fri, 27 Dec 2019 01:32:35 +0000 greenmedinfo 206195 at https://greenmedinfo.com Two case reports of vaccine-induced immune hemolytic anemia. https://greenmedinfo.com/article/two-case-reports-vaccine-induced-immune-hemolytic-anemia PMID:  Transfusion. 2000 Aug ;40(8):907-9. PMID: 10960515 Abstract Title:  Vaccination-associated immune hemolytic anemia in two children. Abstract:  BACKGROUND: Two children in whom acute autoimmune hemolytic anemia (AIHA) developed after vaccination were studied.CASE REPORTS: The children were a 20-month-old girl and a 21-month-old boy. The diagnosis of AIHA was made in accordance with established criteria (hemolysis, positive DAT, and lack of other reasons for the hemolysis). Serologic tests were performed according to standard technique.RESULTS: The girl experienced two attacks of hemolysis. The first episode occurred 2 weeks after oral polio vaccination, and the second episode was observed 7 months later, when she received a simultaneous vaccination against mumps, rubella, and measles. The DAT was strongly positive with anti-C3d. No autoantibodies were detectable in either episode. The boy experienced acute hemolysis a few days after a simultaneous revaccination against diphtheria-pertussis-tetanus, Haemophilus influenzae, hepatitis B, and polio. The DAT using anti-IgG was strongly positive, and the DAT performed with anti-C3d was weakly positive.CONCLUSION: Vaccination-induced AIHA resembles those forms of AIHA related to infectious diseases, and it may occur more frequently than has been reported. <p><a href="https://greenmedinfo.com/article/two-case-reports-vaccine-induced-immune-hemolytic-anemia" target="_blank">read more</a></p> https://greenmedinfo.com/article/two-case-reports-vaccine-induced-immune-hemolytic-anemia#comments Anemia: Hemolytic Vaccine-induced Toxicity Vaccination: All Human: Case Report Sat, 30 Nov 2019 00:13:55 +0000 greenmedinfo 203223 at https://greenmedinfo.com U.S. Physicians Raise Serious Questions About FDA Statements Regarding Teething Products https://greenmedinfo.com/blog/us-physicians-raise-serious-questions-about-fda-statements-regarding-teething-pro <p align="center"><img alt="" src="//cdn.greenmedinfo.com/sites/default/files/ckeditor/blank.justin/images/USPhysiciansRaiseSeriousQuestions.jpg" style="width: 600px; height: 314px;" /></p> <p align="center"><span style="font-size: 22px;"><em><strong>U.S. Physicians Raise Serious Questions About FDA Statements Regarding Teething Products</strong></em></span></p> <p>Washington, D.C.:&nbsp; Physicians trained in integrative medicine today raised concerns that the FDA may have overreacted or worse when issuing warnings about&nbsp;<strong><a href="http://www.greenmedinfo.com/substance/homeopathic-medicine-all" target="_blank">homeopathic</a></strong>&nbsp;teething products earlier this year.</p><p><a href="https://greenmedinfo.com/blog/us-physicians-raise-serious-questions-about-fda-statements-regarding-teething-pro" target="_blank">read more</a></p> https://greenmedinfo.com/blog/us-physicians-raise-serious-questions-about-fda-statements-regarding-teething-pro#comments Anemia: Hemolytic Belladonna Homeopathic Medicine: All Seizures Sudden Infant Death Syndrome (SIDS) Infant and Children's Heatlh corruption in science Homeopathic Vs. Conventional Treatment industry bias Fri, 30 Jun 2017 15:32:46 +0000 GMI reporter 149652 at https://greenmedinfo.com